DIABETES

HEART, PANCREAS and DIABETES TRIUMVIRATE Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucos) levels, it defected from result of insulin secretion, or action, or both. Diabetes mellitus, diabetes referred commonly identified as a disease associated with "sweet urine," and excessive muscle loss in the ancient world. Elevated levels of blood glucose. lead to spillage of glucose into the urine, hence the term sweet urine. Normally insulin a hormone produced by the pancreas, controlled blood glucose levels are tightly. Insulin lowers the blood glucose level. When the blood glucose elevates (for example, after eating food), insulin is released from the pancreas to normalize the glucose level. In patients with diabetes, the absence or insufficient production of insulin causes hyperglycemia. Diabetes is a chronic medical condition, meaning that although it can be controlled, it. lasts a lifetime. Insulin and glucagon is a major hormone in increas and dicreas of glucose blood presure system. It manually active when presure of glucose in our blood increas and dicreas. The metabolisme balance is depend to the maintenance of glucose blood presure close at the point pairs. It is about 90 mg/100 mL in human. Both insulin and glucagon it’s have a big effect to the blood glucose concetration in many way of mechanism. Insulin will decrease the glucose level when the blood glucose getting higher and glucagon will increase the blood glucose level when it getting lower. Glucagon revamped and produced in heart and will activeted just when our body need it. So with healt and normal pancreas and heart we can make a preparation and resistanceable to face and fight diabetes, with cntroled our Food and emotion stability. Stop everything who can involve the diabetes like Smoking, stop get a drunk, eat more fiber food, multiply drinking water and less fatty food A new gen they called enzim Gn-4a glycosyltransferase (GnT-4a) has identificated by Profesor Jamey Marth dan Kazuaki Ohtsubo and his team from the Universitas California, San Diego (UCSD) School of Medicine and the Howard Hughes Medical Institute, it potentially pancreas betha cell stimulated blood glucose to producting insulin with normaly. Research and experiment to a rats showed that high fatty in our blood suppress production enzym GnT-4a it caused pancreas betha cell experiencing shortages in hormone insulin produciton and caused diabetes type 2.